Integrative profiling of alternative splicing induced by U2AF1 S34F mutation in lung adenocarcinoma reveals a mechanistic link to mitotic stress

Medicine and Dentistry

• Stress 100%
• Lung Adenocarcinoma 100%
• Mutant Protein 75%
• Ectopic Expression 75%
• Gene 50%
• Lung Cancer 50%
• Cell Cycle Progression 50%
• Patient 25%
• Development 25%
• Cancer Cell 25%
• Malignant Neoplasm 25%
• Cancer Types 25%
• Carcinogenesis 25%
• Cell Cycle 25%
• Mutant 25%
• Mitosis 25%
• Hematologic Malignancy 25%
• Missense Mutation 25%

Biochemistry, Genetics and Molecular Biology

• Stress 100%
• Mutation 100%
• Alternative Splicing 100%
• Ectopic Expression 60%
• Mutant Protein 60%
• Cell Cycle Progression 40%
• Nested Gene 40%
• Spliceosome 20%
• Development 20%
• Cancer Cell 20%
• Mutant 20%
• Wild Type 20%
• Carcinogenesis 20%
• Mitosis 20%
• Cell Cycle 20%
• Missense Mutation 20%