Inhibiting Mer receptor tyrosine kinase suppresses STAT1, SOCS1/3, and NF-κB activation and enhances inflammatory responses in lipopolysaccharide-induced acute lung injury

Biochemistry, Genetics and Molecular Biology

• STAT1 100%
• Receptor Tyrosine Kinase 100%
• TLR4 100%
• Lipopolysaccharide 100%
• Interleukin-1 50%
• Mediator 50%
• down Regulation 50%
• Tumor Necrosis Factor 50%
• Gelatinase B 50%
• MMP9 50%
• Alveolar Macrophage 50%
• Protein Kinase B 50%

Immunology and Microbiology

• TLR4 100%
• Inflammation Response 100%
• STAT1 100%
• Tyrosine 100%
• Tumor Necrosis Factor 50%
• Interleukin-1 50%
• Blocking Antibody 50%
• Alveolar Macrophage 50%
• Inflammatory Cell 50%
• down Regulation 50%
• Mediator 50%

Pharmacology, Toxicology and Pharmaceutical Science

• Toll Like Receptor 4 100%
• Protein Tyrosine Kinase 100%
• Lipopolysaccharide-Induced Acute Lung Injury 100%
• Inflammation 100%
• Acute Lung Injury 50%
• Gelatinase B 50%
• Tumor Necrosis Factor 50%
• Blocking Antibody 50%